By Andreas Luch
ISBN-10: 3764383372
ISBN-13: 9783764383374
Medical Toxicology is the second one quantity of a three-volume set on molecular, medical and environmental toxicology that provides a finished and in-depth reaction to the expanding value and abundance of chemicals of everyday life. by way of offering interesting insights a ways right down to the molecular point, this three-volume paintings covers the complete variety of recent toxicology with distinctive emphasis on contemporary advancements and achievements. it truly is written for college kids and pros in medication, technological know-how, public wellbeing and fitness or engineering who're hard trustworthy details on poisonous or almost certainly damaging brokers and their adversarial results at the human physique.
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Extra resources for Molecular, Clinical and Environmental Toxicology: Volume 2: Clinical Toxicology (Experientia Supplementum)
Example text
Biochem Biophys Res Commun 380: 76–80 165 Jung N, Haucke V (2007) Clathrin-mediated endocytosis at synapses. Traffic 8: 1129–1136 166 Morgans CW, Kensel-Hammes P, Hurley JB, Burton K, Idzerda R, McKnight GS, Bajjalieh SM (2009) Loss of the synaptic vesicle protein SV2B results in reduced neurotransmission and altered synaptic vesicle protein expression in the retina. PLoS ONE 4: e5230 167 Baldwin MR, Barbieri JT (2009) Association of botulinum neurotoxins with synaptic vesicle protein complexes.
M. Paterson and N. Lima The most convincing evidence of historical reports of mycotoxicosis is a disease that affected many parts of Europe in the tenth century referred to as St Anthony’s or Holy fire, and is considered to have been caused by the consumption of rye contaminated with ergot alkaloids from Claviceps purpurea [2]. , the Ten Plagues of Egypt [3]). Mycotoxins attract worldwide scientific, political and economic attention because of the significant economic losses associated with negative impacts on human health, animal productivity and international trade.
The majority of codon 249 mutations are found in patients with an HBV infection, implicating an association. The mutation only occurred in areas of high AFB1 exposure in comparison to codon 249 mutations in regions of high HBV infection but varying levels of AFB1 exposure. Reproductive effects of aflatoxins AFs are implicated in affecting human males with (a) delayed testicular development, (b) testicular degeneration, (c) decreased reproductive potential, (d) morphological regressive changes in the testis, (e) reduction in size and weight of testis with mild testicular degeneration to complete disappearance of cellular components, (f) decrease in meiotic index, (g) marked decrease in the percentage of live sperm and greater sperm abnormalities, (h) degeneration and desquamation of seminiferous epithelium and decrease in its thickness, (i) decrease in plasma testosterone concentration, and (j) impairment of Leydig cell function [2] – so rather widespread.
Molecular, Clinical and Environmental Toxicology: Volume 2: Clinical Toxicology (Experientia Supplementum) by Andreas Luch
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