By Manfred Bleuler (auth.), William F. Flack Jr., Morton Wiener, Daniel R. Miller (eds.)
William F. Flack, Jr., Daniel R. Miller, and Morton Wiener what's schizophrenia?l This was once the possible basic query posed to a various workforce of investigators requested to give their perspectives at a convention backed by way of the Frances L. Hiatt college of Psychology at Clark collage, Worcester, Massachusetts, in June, 1990. The plan was once to have a small staff of theoretically minded clinicians and investigators from diverse professions and orientations convene to debate and debate conceptual and metatheoretical concerns surrounding "schizophrenia." rather than targeting the most recent empirical findings, we have been basically drawn to having a sequence of exchanges 2 in regards to the very diversified meanings and makes use of of this idea. In our assessment of the literature on schizophrenia, we exposed what looked as if it would us to be a number of, non-overlapping makes use of of the time period. For a few investigators, it seems that for use to specify definite types of humans; for others, it truly is hired to consult sure different types of behaviors. For nonetheless others, the time period is grounded in biochemical occasions, or in socioculturally particular activities. a few choices are explored within the members' papers during this volume.
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Extra resources for What Is Schizophrenia?
The distinction between basic and accessory symptoms was strengthened by the observation that the basic symptoms never disappeared, while accessory symptoms were often subject to change and might disappear intermittently or permanently. Bleuler (1911) and his associate lung (1907) showed convincingly how many of the schizophrenic symptoms could be interpreted in terms of psychoanalytic concepts. lung also regarded schizophrenia as the consequence of an organic brain process. In the overall clinical picture, the accessory, psychologically determined symptoms could be quite dominant; they were, in principle, reversible.
This is, unfortunately, difficult. But we can at least make some negative statements. Autism has nothing to do with the form of dementia that we see in organic brain disorder. Autism does not affect any of the intellectual processes. It is a disturbance of emotional contact with other human beings, but is basically different from what is called autism in children with Kanner's syndrome. If you talk with adult Kanner's patients who are sufficiently intellectually intact, they can assure you that they do want emotional contact with other humans.
Advances in neuropsychiatry and psychopharmacology, vol. 1. New York: Raven Press, pp. 169-178. Stockings, G. (1947). The metabolic brain diseases and their treatment. Baltimore: Williams & Wilkins. R. (1990). Anatomical abnormalities in the brains of monozygotic twins discordant for schizophrenia. The New England Journal of Medicine, 322, 789-794. C. (1978). Genetic and psychosocial factors in schizophrenia: The Finnish adoptive family study. Schizophrenia Bulletin, 13, 477-484. World Health Organization (1979).
What Is Schizophrenia? by Manfred Bleuler (auth.), William F. Flack Jr., Morton Wiener, Daniel R. Miller (eds.)