By Alejandro Berenstein M.D., Pierre Lasjaunias M.D., Ph.D., Karel G. ter Brugge M.D. (auth.)
From the reviews:
"The presentation of the e-book is admirable with such a lot chapters starting with dialogue of simple pathophysiology and carrying on with with scientific features and functional recommendation. … The authors are cautious to not current easily the most recent technological advances, yet to underpin the booklet with clinicopathological points of vascular ailment which they regard as primary for the winning administration of those tough instances. … It features most sensible as a reference textual content and has an available and simple to exploit index which is helping during this regard." (Justin go, Acta Neurochirurgica, 2005)
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Extra resources for Surgical Neuroangiography: Vol. 2: Clinical and Endovascular Treatment Aspects in Adults
Carbon monoxide Carbon monoxide (CO) is believed to serve as a mediator of cerebrovascular relaxation via an increase in CGMB. CO is much less potent than NO as a vasodilator agent, but like NO, may serve multiple neuronal functions. 2. 1 Definition Ischemic stroke is caused by a disruption in blood flow and results in a neurological deficit that persists longer than 24 h. In contrast, transient ischemic attacks (TIAs) are defined as brief episodes of neurological dysfunction resulting from cerebral ischemia.
2000). 7. 8. Stroke risk increases with increasing carotid stenosis. With low-grade stenosis, the risk of stroke is relatively small. Conversely, high-grade stenosis (>75%) carries an annual stroke incidence of approximately 3%. When TIAs are included, the risk increases to approximately 10%, with the majority of events being referable to the ipsilateral stenosed artery (Norris et al. 1991). Carotid stenosis accounts for approximately 30% of CVAs (De Bakey 1996). S. 7. 100 g per minute. Under normal conditions (normoxia and normocapnia), CBF is ample, relative to the metabolic needs of the tissue.
Calcitonin gene-related peptide (CGRP), a potent vasodilator of cerebral vessels, is localized in sensory nerves on the large, medium, and small cerebral vessels. Adrenomedullin is a potent vasodilator of cerebral vessels in vitro and in vivo. Substance P (SP) is the principal member of the tachykinin. SP is primarily localized in the perivascular sensory nerves, where it induces vasodilatation (although there were some exceptions to this effect). SP may be involved in vasodilatation and plasma extravasation in the extracerebral circulation and thereby mediates neurogenic inflammation.
Surgical Neuroangiography: Vol. 2: Clinical and Endovascular Treatment Aspects in Adults by Alejandro Berenstein M.D., Pierre Lasjaunias M.D., Ph.D., Karel G. ter Brugge M.D. (auth.)