By Mark Hallett, Werner Poewe
ISBN-10: 0470066482
ISBN-13: 9780470066485
Over the last few a long time, neurology has noticeable astonishing advancements in diagnostic innovations, really in neuroimaging and molecular genetics, observed by way of increasingly more powerful cures to regard neurological sickness. this can be fairly real for the sphere of stream problems. because the hugely profitable advent of levodopa approximately forty years in the past, the healing ideas for Parkinson's Read more...
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Additional resources for Therapeutics of Parkinson's disease and other movement disorders
Example text
And Halliwell, B. (1997) A generalised increase in protein carbonyls in the brain in Parkinson’s but not incidental Lewy body disease. Journal of Neurochemistry, 69 (3), 1326--1329. , Jenner, P. (Sep 1997) A generalised increase in protein carbonyls in the brain in Parkinson’s but not incidental Lewy body disease. Journal of Neurochemistry, 69 (3), 1326--1329. F. A. (2003) Inhibition of proteasomal activity causes inclusion formation in neuronal and non-neuronal cells overexpressing parkin. Molecular Biology of the Cell, 14 (11), 4541--4556.
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2003) Staging of brain pathology related to sporadic Parkinson’s disease. Neurobiology of Aging, 24 (2), 197--211. M. A. (2002) Hydroxynonenal adducts indicate a role for lipid peroxidation in neocortical and brainstem Lewy bodies in humans. Neuroscience Letters, 319 (1), 25--28. Caughey, B. T. (2003) Protofibrils, pores, fibrils, and neurodegeneration: separating the responsible protein aggregates from the innocent bystanders. Annual Review of Neuroscience, 26, 267--298. , Ilijic, E. et al. (2007) “Rejuvenation” protects neurons in mouse models of Parkinson’s disease.
Therapeutics of Parkinson's disease and other movement disorders by Mark Hallett, Werner Poewe
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